https://nova.newcastle.edu.au/vital/access/ /manager/Index en-au 5 Melatonin prevents postovulatory oocyte aging in the mouse and extends the window for optimal fertilization in vitro https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:20252 Sat 24 Mar 2018 07:59:55 AEDT ]]> Functional deletion of Txndc2 and Txndc3 increases the susceptibility of spermatozoa to age-related oxidative stress https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:19972 Sat 24 Mar 2018 07:54:28 AEDT ]]> The presence of a truncated base excision repair pathway in human spermatozoa that is mediated by OGG1 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:20027 Sat 24 Mar 2018 07:50:55 AEDT ]]> The source and significance of DNA damage in human spermatozoa; a commentary on diagnostic strategies and straw man fallacies https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:20028 Sat 24 Mar 2018 07:50:55 AEDT ]]> Oxidative stress, spermatozoa and leukocytic infiltration: relationships forged by the opposing forces of microbial invasion and the search for perfection https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:20031 Sat 24 Mar 2018 07:50:54 AEDT ]]> The senescence-accelerated mouse prone 8 as a model for oxidative stress and impaired DNA repair in the male germ line https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:20025 Sat 24 Mar 2018 07:50:54 AEDT ]]> On methods for the detection of reactive oxygen species generation by human spermatozoa: analysis of the cellular responses to catechol oestrogen, lipid aldehyde, menadione and arachidonic acid https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:20026 Sat 24 Mar 2018 07:50:51 AEDT ]]> Electrophilic aldehydes generated by sperm metabolism activate mitochondrial reactive oxygen species generation and apoptosis by targeting succinate dehydrogenase https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:22207 R²= 0.89) in untreated populations of human spermatozoa emphasized the pathophysiological significance of these findings. The latter also provide a biochemical explanation for the self-perpetuating nature of oxidative stress in the male germ line, with the products of lipid peroxidation stimulating free radical generation by the sperm mitochondria in a positive feedback loop.]]> Sat 24 Mar 2018 07:11:16 AEDT ]]>